index - Organisation de la cellule musculaire et thérapie de la myopathie centronucléaire autosomique dominante Accéder directement au contenu

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AAV8 Dystrophie musculaire d'Emery Dreifuss Cellules de crête neurale Clathrine Caveolins Animal models of human disease Disease heterogeneity Muscle Cavéoles BAR proteins Adhesion Dystrophin Allele-specific silencing AAV CTL Migration Cell migration Dynamin overexpression Congenital myopathy RNA interference Biomarkers Caveolae DNM2 Correlative microscopy Muscular dystrophy Developmental myosin heavy chain Domaine LEM Nesprin Dynamine Neural crest cells Cell signaling Cavins Cancer ACTN2 Duchenne muscular dystrophy DMD Duchenne muscular dystrophy Allele‐specific silencing therapy Clathrin Dullard Myopathie AFM Cross-bridge kinetics Nucleus CAV-3 gene Endocytosis Autosomal dominant centronuclear myopathy Alpha-actinin-2 Actin nucleus Lamin Cytoskeleton Dystrophie musculaire de Duchenne Coeur Atrial cardiac defects A-type lamins Satellite cell Disease modifiers Cross-presentation Skeletal muscle Skin Dynamin 2 Autophagy Becker muscular dystrophy BMD Centronuclear myopathy Adeno-Associated virus BAF Diaphragm Myopathy Mechanotransduction Allele specific RNA interference Antisense oligonucleotides Core myopathy Nuclear envelope Allele-specific silencing therapy Cellular neuroscience Dominant centronuclear myopathy Amphiphysin Autophagosome maturation Atrial heart defects Autophagosome Myosin Cardiotoxin Dynamin Gene therapy Adult patients Developmental biology BMP signaling Actin Duchenne Muscular Dystrophy Cytosquelette Ctdnep1 Cardiomyopathies Adeno-associated virus Charcot-Marie-Tooth Biophysics AD-CNM Caveolin Outflow tract Adeno-associated virus vector DMyHC Cell proliferation